Bilateral abducens palsies and facial weakness as initial manifestations of a Chiari 1 malformation.
Optom Vis Sci. 2007 Oct;84(10):936-40
Authors: Pilon A, Rhee P, Newman T, Messner L
PURPOSE: Chiari I malformations are rare, congenital anomalies involving the caudal herniation of the cerebellar tonsils into the upper cervical spinal canal. Osseous abnormalities of the skull permit the extension of the hindbrain below the foramen magnum and engender syringohydromyelia formation. Neuroophthalmic manifestations are not uncommon; nevertheless, this is the first report of concomitant bilateral abducens palsies and facial nerve impairment as presenting manifestations of a Chiari I malformation. CASE REPORT: A 30-year-old black female presented with a recent history of blurred vision and binocular diplopia in both left and right gazes. Headaches, dizziness, and orofacial sensorimotor impairments were noted in conjunction with the onset of her diplopia. No history of surgery, trauma, or systemic illness was elicited. Extraocular motility testing confirmed a complete bilateral abduction deficit. Additional cranial nerve testing revealed bilateral facial nerve weakness and tactile hypesthesia in the perioral region. No taste disturbances were reported. Nuclear magnetic resonance imaging studies revealed a Chiari I malformation with a syringohydromyelia formation in the vicinity of the patient's sixth cervical vertebrae. Foramen magnum decompression surgery was performed. Two months after surgery, abduction capacity was restored along with resolution of symptoms of perioral numbness and signs of facial weakness. CONCLUSION: Bilateral sixth nerve palsies are rare clinical commodities especially when they do not occur in isolation. Intracranial neoplams, trauma, subarachnoid hemorrhaging, demyelinating disease, and meningeal infarcts have all been shown to induce mixed cranial neuropathies. The concomitant bilateral abducens palsy and concomitant facial nerve weakness reported in this case represents an atypical and previously unreported presentation of a Chiari I malformation.
PMID:- Face weakness causes 18049357 [PubMed - indexed for MEDLINE ]
Magnetic resonance imaging changes in synchronous bilateral progressive facial nerve weakness.
J Thorac Oncol. 2006 Jun;1(5):487-8
Authors: Fogarty GB, Cassumbhoy R, Ball D
PMID:- Face weakness causes 17409903 [PubMed - indexed for MEDLINE ]
Facial weakness in a haemodialysis patient.
Lancet. 2007 Feb 24;369(9562):714
Authors: Papadakis M, Fee P, Wilhelm T, Davenport A, Connolly J
PMID:- Face weakness causes 17321322 [PubMed - indexed for MEDLINE ]
On call. Last month I developed severe weakness on the whole left side of my face. My doctor diagnosed Bell's palsy and referred me to a neurologist. I recovered before I got to see her, so I didn't keep the appointment. Although my face is now back to normal, I'd like to know more about the condition. What can you tell me?
Harv Mens Health Watch. 2006 Nov;11(4):8
Authors: Simon HB
PMID:- Face weakness causes 17278299 [PubMed - indexed for MEDLINE ]
I cannot smile or wink anymore: facial nerve weakness after acoustic neuroma surgery.
Curr Surg. 2005 Mar-Apr;62(2):156-61; quiz 161
Authors: Trang TT, Megerian CA
PMID:- Face weakness causes 15796934 [PubMed]
Aneurysmal expansion presenting as facial weakness: case report and review of the literature.
Authors: Neimat JS, Hoh BL, McKenna MJ, Rabinov JD, Ogilvy CS
OBJECTIVE AND IMPORTANCE: Facial paralysis and hemifacial spasm are rare presentations of aneurysms in the posterior fossa. We report an unusual case of rapidly progressive facial palsy caused by the acute expansion of an arteriovenous malformation-associated anteroinferior cerebellar artery aneurysm. The case is notable for the rapid progression of symptoms and their precise correlation with radiographic changes, emphasizing the potential dynamic nature of aneurysms associated with arteriovenous malformations. CLINICAL PRESENTATION: A 56-year-old woman with severe headache and nausea was seen in a local emergency room, where she underwent a neurological examination with unremarkable results and a head computed tomographic scan demonstrating acute hemorrhage in the ambient cisterns. Conventional and computed tomographic angiograms demonstrated an arteriovenous malformation in the right cerebellopontine angle fed by the anteroinferior cerebellar and superior cerebellar arteries. A micro-aneurysm measuring 3 mm was noted within the internal carotid artery on the meatal loop of the anteroinferior cerebellar artery. Two weeks later, a rapidly progressive right facial weakness developed in the patient, progressing to complete facial plegia over 12 hours, and complete sensory neural hearing loss. Repeat angiography demonstrated expansion of the previously visualized aneurysm to 8 x 4 mm. INTERVENTION: The patient was taken to surgery for clipping of the aneurysm, which required petrous drilling to unroof the canal. She has experienced substantial recovery of facial nerve function. CONCLUSION: Although compression of the VIIth-VIIIth nerve complex is an unusual presentation for posterior fossa aneurysms, it represents an important potential complication of vascular pathological features. The rapid aneurysmal expansion, confirmed by imaging and correlating with the rapid onset of symptoms, gives an impressive demonstration of the anatomic changes that can occur in an aneurysm associated with an arteriovenous malformation.
PMID:- Face weakness causes 15617605 [PubMed - indexed for MEDLINE ]
Pathologic quiz case: a 42-year-old man with right facial swelling and weakness. Dedifferentiated acinic cell carcinoma of the parotid gland.
Arch Pathol Lab Med. 2004 Mar;128(3):e52-3
Authors: Schultz AM, Thomas AB, Henley JD, Badve S
PMID:- Face weakness causes 14987131 [PubMed - indexed for MEDLINE ]
Pathologic quiz case: a child with facial and proximal limb weakness.
Arch Pathol Lab Med. 2003 Jun;127(6):755-6
Authors: Mitra S, Prayson RA, Friedman NR
PMID:- Face weakness causes 12741907 [PubMed - indexed for MEDLINE ]
Chronic asymmetric progressive external ophthalmoplegia with right facial weakness: a unique presentation of mitochondrial myopathy.
J Neurol Neurosurg Psychiatry. 2002 Jul;73(1):95
Authors: Sharma NK, Gujrati M, Kumar J, Kattah JC
PMID:- Face weakness causes 12082069 [PubMed - indexed for MEDLINE ]
Limb girdle and facial weakness in female carriers of X-linked myotubular myopathy mutations.
Neurology. 2001 Sep 11;57(5):900-2
Authors: Sutton IJ, Winer JB, Norman AN, Liechti-Gallati S, MacDonald F
Although X-linked myotubular myopathy (XLMTM) is a recessive disorder, heterozygous female carriers of MTM1 mutations may present with limb girdle and facial weakness. It is proposed that manifesting heterozygote females with XLMTM have a skewed pattern of X-chromosome inactivation. However, skewed X-chromosome inactivation was not detected in either the lymphocyte or muscle DNA of a woman who presented with limb girdle/facial weakness and was found to be heterozygous for the R224X mutation.
PMID:- Face weakness causes 11552027 [PubMed - indexed for MEDLINE ]
Facial electromyography in newborn and young infants with congenital facial weakness.
Dev Med Child Neurol. 2001 Jun;43(6):421-7
Authors: Renault F
PMID:- Face weakness causes 11409833 [PubMed - indexed for MEDLINE ]
Facial and skeletal malformations, mental retardation, aganglionosis, and neurogenic muscle weakness: a variant of Niikawa-Kuroki syndrome or a new syndrome?
J Child Neurol. 2001 Apr;16(4):296-8
Authors: Greco D, Romano C, Elia M
We report a 10-year-old boy with multiple congenital anomalies/mental retardation syndrome, who also presented with aganglionosis and neurogenic muscle weakness. Some phenotypic manifestations of our patient overlap with those observed in the Niikawa-Kuroki syndrome; however, the hypothesis of a new distinct entity, with simultaneous involvement of the central and peripheral nervous system, is considered.
PMID:- Face weakness causes 11332467 [PubMed - indexed for MEDLINE ]
One and one-half syndrome with supranuclear facial weakness: magnetic resonance imaging localization.
Arch Neurol. 1999 Dec;56(12):1509-11
Authors: Anderson CA, Sandberg E, Filley CM, Harris SL, Tyler KL
OBJECTIVE: To provide clinicoanatomical correlation for a small pontine tegmental ischemic stroke producing the one and one-half syndrome associated with supranuclear facial weakness. DESIGN: Case report. SETTING: Tertiary care center. PATIENT: A 70-year-old man developed left-sided facial weakness sparing the forehead, a left internuclear ophthalmoplegia, and a complete left horizontal gaze palsy immediately after percutaneous transluminal coronary angioplasty. Magnetic resonance imaging demonstrated a small lesion in the left paramedian aspect of the dorsal pontine tegmentum. MAIN OUTCOME AND RESULTS: Electromyographic findings were consistent with supranuclear facial involvement. The patient had nearly complete recovery after 1 year. CONCLUSIONS: To our knowledge, this is the first report of supranuclear facial weakness in association with the one and one-half syndrome. The location of the lesion provides evidence of the existence of corticofugal fibers that extend to the facial nucleus in the dorsal paramedian pontine tegmentum.
PMID:- Face weakness causes 10593308 [PubMed - indexed for MEDLINE ]
Ipsilateral facial weakness in upper medullary infarction-supranuclear or infranuclear origin?
J Neurol. 1999 Sep;246(9):798-801
Authors: Urban PP, Wicht S, Fitzek S, Marx J, Thömke F, Fitzek C, Hopf HC
We describe two patients with upper medullary infarctions showing ipsilateral facial weakness and relative sparing of the upper facial muscles. Electrophysiological follow-up using transcranial magnetic stimulation of the motor cortex in combination with stimulation of the peripheral facial nerve disclosed a supranuclear (corticofacial) tract lesion in one patient and a partial nuclear/infranuclear intra-axial facial nerve lesion in another.
PMID:- Face weakness causes 10525977 [PubMed - indexed for MEDLINE ]
Blink reflex recovery in facial weakness: an electrophysiologic study of adaptive changes.
Neurology. 1999 Mar 10;52(4):834-8
Authors: Syed NA, Delgado A, Sandbrink F, Schulman AE, Hallett M, Floeter MK
OBJECTIVE: To study the electrophysiologic effects of unilateral facial weakness on the excitability of the neuronal circuitry underlying blink reflex, and to localize the site of changes in blink reflex excitability that occur after facial weakness. BACKGROUND: Eyelid kinematic studies suggest that adaptive modification of the blink reflex occurs after facial weakness. Such adaptations generally optimize eye closure. A report of blepharospasm following Bell's palsy suggests that dysfunctional adaptive changes can also occur. METHODS: Blink reflex recovery was evaluated with paired stimulation of the supraorbital nerve at different interstimulus intervals. Comparisons were made between normal control subjects and patients with Bell's palsy who either recovered facial strength or who had persistent weakness. RESULTS: Blink reflex recovery was enhanced in patients with residual weakness but not in patients who recovered facial strength. Facial muscles on weak and unaffected sides showed enhancement. In patients with residual weakness, earlier blink reflex recovery occurred when stimulating the supraorbital nerve on the weak side. Sensory thresholds were symmetric. CONCLUSION: Enhancement of blink reflex recovery is dependent on ongoing facial weakness. Faster recovery when stimulating the supraorbital nerve on the paretic side suggests that sensitization may be lateralized, and suggests a role for abnormal afferent input in maintaining sensitization. Interneurons in the blink reflex pathway are the best candidates for the locus of this plasticity.
PMID:- Face weakness causes 10078735 [PubMed - indexed for MEDLINE ]
Delayed facial weakness after microvascular decompression of cranial nerve VII.
Surg Neurol. 1998 Nov;50(5):449-52
Authors: Lovely TJ, Getch CC, Jannetta PJ
BACKGROUND: Retromastoid craniectomy and microvascular decompression of cranial nerve VII for hemifacial spasm is a well accepted and effective treatment. Risks of the operation relate to the surgical approach in general and to the seventh nerve in particular. Delayed facial weakness is an unusual and little-described complication of the procedure. The purpose of this review is to describe this complication and the characteristics of the patients so affected. METHODS: Between 1972 and 1996, 985 patients have undergone microvascular decompression for hemifacial spasm. During this time, 28 patients (2.8%) undergoing decompression of the facial nerve and 1 patient undergoing decompression of the cochlear nerve for tinnitus developed delayed facial palsy. RESULTS: The weakness was at least a House Grade III or worse and was complete in 11 of the patients. The time to occurrence averaged 12 days, with a tight range of 7 to 16 days. There were no factors such as duration of symptoms, intraoperative findings, or preoperative botulinum injections that were predictive of this postoperative weakness. In all patients there was almost complete recovery (House Grade I or II). CONCLUSIONS: Delayed facial weakness after MVD of CN VII can occur in up to 3% of cases. The onset of weakness after operation is consistent in its timing, occurring on average 12 days after the procedure. Although the etiology of this complication is uncertain, the palsy spontaneously resolves with a good or excellent outcome.
PMID:- Face weakness causes 9842870 [PubMed - indexed for MEDLINE ]
Facial weakness in hereditary inclusion body myopathies.
Neurology. 1998 Jun;50(6):1925-6
Authors: Argov Z, Sadeh M, Eisenberg I, Karpati G, Mitrani-Rosenbaum S
PMID:- Face weakness causes 9633772 [PubMed - indexed for MEDLINE ]
Images in clinical medicine. Volitional and emotional supranuclear facial weakness.
N Engl J Med. 1998 May 21;338(21):1515
Authors: Ross RT, Mathiesen R
PMID:- Face weakness causes 9593790 [PubMed - indexed for MEDLINE ]
Case of the month: June 1997--a 42 year old man with left facial weakness.
Brain Pathol. 1998 Jan;8(1):233-4
Authors: DeRubeis DA, Woulfe J, Rosso D, Lownie S, Parnes L, Lee D, Strong MJ, Hammond RR
A 42 yr old male presented with left facial weakness. MRI showed lesions affecting the distal seventh nerve and third division of the trigeminal nerve. The seventh nerve was biopsied and showed a malignant epithelioid schwannoma. The patient underwent extensive resection followed by irradiation. This is one of very few examples of intracranial malignant peripheral nerve sheath tumors and the first reported example of an intracranial malignant epithelioid schwannoma. The literature is reviewed and completeness of resection appears to be the most pertinent prognostic factor.
PMID:- Face weakness causes 9458180 [PubMed - indexed for MEDLINE ]
Central facial weakness due to medial medullary infarction: the course of facial corticobulbar fibres.
J Neurol Neurosurg Psychiatry. 1997 Sep;63(3):391-3
Authors: Terao S, Takatsu S, Izumi M, Takagi J, Mitsuma T, Takahashi A, Takeda A, Sobue G
Two patients are reported with contralateral hemiparesis including a face of supranuclear type, caused by an infarct of the unilateral ventromedial part of the upper medulla. Data from these patients support the hypothesis that part of the corticobulbar fibres supplying the lower facial muscles descend ipsilaterally in the ventromedial part of the upper medulla and then, after decussation, ascend rostrally to the contralateral facial nucleus.
PMID:- Face weakness causes 9328262 [PubMed - indexed for MEDLINE ]
Gingivitis, facial weakness and focal seizures.
Postgrad Med J. 1997 Jun;73(860):327-8
Authors: Lee PY, Hillier CE, Viagappan GM
PMID:- Face weakness causes 9246327 [PubMed - indexed for MEDLINE ]
Dejerine-Sottas disease with sensorineural hearing loss, nystagmus, and peripheral facial nerve weakness: de novo dominant point mutation of the PMP22 gene.
J Med Genet. 1996 Dec;33(12):1048-9
Authors: Ionasescu VV, Searby C, Greenberg SA
A 32 year old woman with Dejerine-Sottas disease and negative family history is reported. Clinical onset of her condition was with congenital weakness of her distal four extremities, accompanied by peripheral facial nerve weakness, deafness, and nystagmus. She has used a wheelchair all her life. Sural nerve biopsy showed proliferation of Schwann cells, extensive endoneural fibrosis, axon loss, and demyelination. MNCVs showed marked slowing. MRI of the brain was normal. Molecular genetic studies indicated a de novo dominant missense point mutation of exon 3 of the peripheral myelin protein 22 gene at nucleotide 264 causing replacement of serine with leucine.
PMID:- Face weakness causes 9004143 [PubMed - indexed for MEDLINE ]
Facial weakness and rash. Ramsay Hunt syndrome (herpes zoster cephalicus, herpes zoster oticus, herpes zoster auricularis).
Acad Emerg Med. 1996 Dec;3(12):1144-5, 1153-5
Authors: Birinyi F
PMID:- Face weakness causes 8959171 [PubMed - indexed for MEDLINE ]
[Central facial weakness due to medullary pyramidal infarction; a case report]
Rinsho Shinkeigaku. 1996 Nov;36(11):1259-61
Authors: Terao S, Takatsu S, Izumi M, Mitsuma T, Sobue G
We reported a hypertensive 40-year-old man who developed sudden right hemiparesis, deep sensory disturbance, left hypoglossal nerve palsy, and mild right central facial weakness. MRI of the brain showed an infarct located in the left upper medullary pyramid. Course and connection of the corticobulbar pathway are not well known in human. Recently Cavazos (1996) described a hypothesis that some facial corticobulbar fibers descend caudally ipsilaterally as low as the upper medulla, making a loop before decussating and ascending to the contralateral facial nucleus Contralateral central facial weakness may result from interruption of these descending fibers.
PMID:- Face weakness causes 9046860 [PubMed - indexed for MEDLINE ]
Cases from the aerospace medicine resident's teaching file #65. Complicated diagnosis of left ear pain and facial weakness in an aviator.
Aviat Space Environ Med. 1996 Aug;67(8):787-9
Authors: O'Brien DM
Cases from the aerospace medicine resident's teaching file #65. Complicated diagnosis of left ear pain and facial weakness in an aviator.
PMID:- Face weakness causes 8853838 [PubMed - indexed for MEDLINE ]
Case records of the Massachusetts General Hospital. Weekly clinicopathological exercises. Case 16-1996. A 36-year-old woman with bilateral facial and hand weakness and impaired truncal sensation.
N Engl J Med. 1996 May 23;334(21):1389-94
PMID:- Face weakness causes 8614426 [PubMed - indexed for MEDLINE ]
Botulinum-induced facial weakness impairs communication.
Mov Disord. 1996 Jan;11(1):101-2
Authors: Kaufman DM, Abraham S
PMID:- Face weakness causes 8771078 [PubMed - indexed for MEDLINE ]
Juvenile myasthenia gravis with predominant facial weakness in a 7-year-old boy.
Int J Pediatr Otorhinolaryngol. 1995 May;32(2):167-9
Authors: Kini PG
Myasthenia gravis is the most prevalent of the diseases of the neuromuscular junction in children. The most common clinical finding is ptosis, although ophthalmoplegia and facial weakness are commonly present. This paper reports juvenile myasthenia gravis in a 7-year-old boy with predominant facial muscle weakness without ophthalmoplegia and ptosis. This was detected post-operatively after adenotonsillectomy, as there was severe respiratory distress. Prompt diagnosis and treatment was life saving. A CT scan of the chest revealed thymoma. After the surgical removal of the thymoma, the child has been followed up for a year and is asymptomatic.
PMID:- Face weakness causes 7657471 [PubMed - indexed for MEDLINE ]
An infarcted Warthin's tumour presenting with facial weakness.
Br J Oral Maxillofac Surg. 1993 Oct;31(5):311-2
Authors: Newman L, Loukota RA, Bradley PF
It is rare for benign salivary gland neoplasms to cause facial nerve weakness. We present such a case occurring in a 70-year-old patient with an infarcted Warthin's tumour and review the benign causes of facial nerve impairment.
PMID:- Face weakness causes 8218086 [PubMed - indexed for MEDLINE ]
Functional facial nerve weakness after surgery for benign parotid tumors: a multivariate statistical analysis.
Head Neck. 1993 Mar-Apr;15(2):147-52
Authors: Mra Z, Komisar A, Blaugrund SM
A retrospective study was done on 64 patients who underwent superficial or subtotal parotidectomy for a primary benign tumor of the parotid gland. Factors, such as age, sex, smoking, alcohol consumption, type of surgery, duration of surgery, pathology of lesion, and size of lesion, were reviewed in a multivariate statistical analysis to determine if any factor alone or in combination contributed to the development of functional facial nerve weakness postoperatively. Only the age of the patient was found to have a statistically significant causal relation using the Pearson chi-square method (p = 0.015). The marginal mandibular branch was affected in nine of 10 cases. Different surgical approaches cited in the literature are discussed, along with the possible role of ischemic injury to the facial nerve during parotidectomy.
PMID:- Face weakness causes 8382671 [PubMed - indexed for MEDLINE ]
Facial weakness. A comparison of clinical and photographic methods of observation.
Arch Otolaryngol Head Neck Surg. 1991 Aug;117(8):906-9
Authors: Smith IM, Murray JA, Cull RE, Slattery J
The search for an internationally acceptable facial grading system has resulted in an assessment of existing methods by several investigators. These studies were based on observations of video film taken of patients with varying degrees of facial malfunction. Although the grading systems were evaluated, the use of videotape has never been compared with clinical examination and its suitability for this type of work is, therefore, unknown. We used nine facial grading systems to compare the results of clinical observation with those of photographic methods of presentation. The latter included videotape, photographic slides, and a combination of the two. The correlation between clinical examination findings and findings of any of the photographic methods was poor, suggesting the need for a standard form of presentation when grading patients. The most consistent results were found with either clinical examination or photographic slides; videotape was the least reliable.
PMID:- Face weakness causes 1892624 [PubMed - indexed for MEDLINE ]
A 44-year-old man with erythema of the face, neck, and chest, weakness, and palpitations.
J Emerg Nurs. 1991 Apr;17(2):116-7
Authors: Carson L
PMID:- Face weakness causes 2010958 [PubMed - indexed for MEDLINE ]
Distinguishing and improving dysarthria due to facial weakness.
Arch Neurol. 1989 Feb;46(2):125
Authors: Starr A
PMID:- Face weakness causes 2916949 [PubMed - indexed for MEDLINE ]
Investigation of unilateral facial weakness: magnetic stimulation of the proximal facial nerve and of the face-associated motor cortex.
J Neurol. 1989 Feb;236(2):102-7
Authors: Meyer BU, Britton TC, Benecke R
Twenty-four patients with unilateral facial weakness of various aetiologies were investigated using a magnetic stimulator to stimulate the proximal segment of the facial nerve directly (short latency response) and also to activate the facial motoneurons bilaterally via corticonuclear pathways by placing the stimulating coil over the motor cortex (long latency responses). Electromyographic recordings were taken from both mentalis muscles using concentric needle electrodes. Seventeen patients were investigated at various times after onset of idiopathic facial palsy (Bell's palsy). In the acute stage (less than 5 days after onset) short and long latency responses on the paretic side were abnormal, being absent in all but one patient, in whom the short latency response was delayed. These abnormal responses were the earliest neurographic correlate for nerve conduction block. In 4 out of 9 patients seen up to 30 days after onset of palsy, trans-synaptically evoked long latency responses were absent. In patients examined more than 2 months after onset, long latency responses could always be obtained and, in 5 of 8 patients, short latency responses could also be elicited, indicating a return of the direct excitability of the nerve. Five patients with cerebral hemisphere lesions causing mild unilateral facial weakness had absent long latency responses when stimulating over the affected hemisphere, but normal bilateral long latency responses following stimulation over the unaffected cerebral hemisphere; short latency responses were normal. Magnetic stimulation of the brain and of the facial nerve can differentiate between central and peripheral causes of unilateral facial weakness and may prove useful in the early assessment of the degree of conduction block in Bell's palsy.
PMID:- Face weakness causes 2709050 [PubMed - indexed for MEDLINE ]
Distinguishing and improving dysarthria due to facial weakness.
Arch Neurol. 1988 Oct;45(10):1061
Authors: Starr A
PMID:- Face weakness causes 3178525 [PubMed - indexed for MEDLINE ]
Facial weakness without ocular weakness in myasthenia gravis.
Muscle Nerve. 1988 Feb;11(2):185-6
Authors: Chia LG
PMID:- Face weakness causes 3343996 [PubMed - indexed for MEDLINE ]
A case of generalized myasthenia gravis presenting predominant bilateral facial weakness without ocular muscle involvement.
Taiwan Yi Xue Hui Za Zhi. 1987 Nov;86(11):236-8
Authors: Chia LG
PMID:- Face weakness causes 3443843 [PubMed - indexed for MEDLINE ]
Facial weakness from unknown childhood cause: management dilemma.
Am J Otol. 1986 Sep;7(5):393-4
Authors: Miller GW
PMID:- Face weakness causes 3789126 [PubMed - indexed for MEDLINE ]
Double internal auditory canal associated with progressive facial weakness.
Am J Otol. 1986 Jul;7(4):275-81
Authors: Curtin H, May M
A case of progressive facial weakness associated with an anomaly of the internal auditory canal and labyrinthine segment of the facial nerve is documented with clinical, radiological, and surgical findings. Each portion of a double internal auditory canal (aberrant facial nerve canal) was found to contain a portion of the facial nerve, and each portion of the facial nerve was explored because a tumor was suspected to be the cause of the progressive weakness. However, no mass was found. The embryologic factors that may lead to such an abnormality and the pathophysiologic mechanisms that may have resulted in the facial weakness are discussed.
PMID:- Face weakness causes 3740236 [PubMed - indexed for MEDLINE ]
Familial myopathies with restricted distribution, facial weakness and inflammatory changes in affected muscles.
J Neurol. 1985;231(6):295-300
Authors: Bacq M, Telerman-Toppet N, Coërs C
A myopathy characterized by restricted involvement of few muscles and inflammatory cell infiltration was observed in three families. In the first family, clinical features, hereditary transmission and biopsy findings were consistent with the diagnosis of facioscapulohumeral dystrophy. However in three of the four affected members, the occurrence of atrophies was specifically initiated by severe muscular pain. In the second family two 8-year-old identical twins had both marked facial weakness and atrophy limited to the right quadriceps femoris. In the third family, marked asymmetry of muscular wasting in the upper limbs was found in the 17-year-old daughter of a man suffering from facial and axial weakness. The indication of corticotherapy in such cases is discussed.
PMID:- Face weakness causes 3973638 [PubMed - indexed for MEDLINE ]
Facial weakness and oligosyndactyly: ? independent variable features of familial type of the Möbius syndrome.
Clin Genet. 1983 Nov;24(5):350-4
Authors: Mitter NS, Chudley AE
A family consisting of two daughters, one with an isolated oligosyndactyly and the other with the Möbius Syndrome (VIth and VIIth nerve dysplasia) is presented. The majority of the individuals previously reported with an association of occulo-facial diplegia and limb anomalies have been sporadic. However, on examination of the parents in the family we report, the mother was found to have bilateral facial weakness. Isolated limb anomalies may, therefore, be a variable expression of a broad spectrum type of the Möbius Syndrome, with an autosomal dominant mode of inheritance.
PMID:- Face weakness causes 6652945 [PubMed - indexed for MEDLINE ]
Case records of the Massachusetts General Hospital. Weekly clinicopathological exercises. Case 45-1982. A 63-year-old woman with hypercalcemia and facial weakness.
N Engl J Med. 1982 Nov 11;307(20):1257-64
PMID:- Face weakness causes 7133056 [PubMed - indexed for MEDLINE ]
New test methods for estimation of facial weakness.
Tohoku J Exp Med. 1973 Apr;109(4):377-83
Authors: Hozawa J, Sasaki Y, Takahashi S, Kusakari J
PMID:- Face weakness causes 4722251 [PubMed - indexed for MEDLINE ]
Cardio-facial syndrome (Cayler's syndrome). Congenital heart disease and facial weakness. A report on two cases.
Cent Afr J Med. 1972 Aug;18(8):162
Authors: Kendall HM
PMID:- Face weakness causes 5080378 [PubMed - indexed for MEDLINE ]
A sensitive sign of facial nerve weakness.
Laryngoscope. 1972 Jan;82(1):17-20
Authors: Stroud MH, Thalmann R
PMID:- Face weakness causes 5008757 [PubMed - indexed for MEDLINE ]
Cardiofacial syndrome. Report of two cases o congenital heart disease with congenital unilateral partial facial weakness.
Indian J Pediatr. 1971 Oct;38(285):396-8
Authors: Mahajan CM, Bhattacharjee S, Bidwai PS
PMID:- Face weakness causes 5143561 [PubMed - indexed for MEDLINE ]
Congenital cardiac malformation and facial weakness.
Br Med J. 1969 Jul 19;3(5663):127
PMID:- Face weakness causes 5792907 [PubMed - indexed for MEDLINE ]
Cardiofacial syndrome. Congenital heart disease and facial weakness, a hitherto unrecognized association.
Arch Dis Child. 1969 Feb;44(233):69-75
Authors: Cayler GG
PMID:- Face weakness causes 5765991 [PubMed - indexed for MEDLINE ]
Cardiofacial syndrome. Report of two cases of congenital heart disease with congenital unilateral partial facial weakness.
C R Seances Soc Biol Fil. 1969;163(5):1149-52
Authors: Bastide P, Couquelet J, Couquelet J
PMID:- Face weakness causes 4335332 [PubMed - indexed for MEDLINE ]
Fluctuating and intermittent facial weakness following a local anaesthetic.
J Neurol Neurosurg Psychiatry. 1966 Aug;29(4):367-70
Authors: Kinsbourne M, Rushworth G
PMID:- Face weakness causes 5969095 [PubMed - indexed for MEDLINE]